What It’s Like To Live With Tuberculosis In The United States

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  • By Lauren Weber The Morning Email Editor, The Huffington Post

In August 2014, Kate O’Brien, a 34-year-old media producer from Brooklyn, found out she was expecting her second child.

She was ecstatic. But this pregnancy didn’t proceed like the first. For the next few months, O’Brien had a cold she couldn’t shake. She woke up in the middle of the night drenched in sweat. She wanted to blame it on her pregnancy, yet she kept losing weight.

She could barely eat. She coughed up balls of bloody mucus. Her throat burned. None of her doctors could figure out what was wrong.

A physician sent her to Mount Sinai West Hospital in Manhattan in January 2015, when, at five months pregnant, she still couldn’t gain any weight.

“No one likes a skinny pregnant lady,” she said.

O’Brien expected to stay at the hospital overnight. She didn’t get a chance to say goodbye to her 2-year-old, Donny, but she figured she’d be home soon.

She didn’t walk out of the hospital for 75 days.

The doctors at Mount Sinai diagnosed O’Brien with infectious tuberculosis. After a few days in the intensive care unit, she was shifted to a negative-pressure isolation room, which helps contain the infected air. Signs announcing “WARNING: Infectious Disease” were affixed to the room’s airtight set of double doors. And all O’Brien could think about was what this meant for her unborn baby.

The federal policy that governs medical isolation and quarantine in the U.S. applies to just a handful of diseases. Most of them, such as cholera, smallpox and the plague, are vanishingly rare in the U.S. But tuberculosis is not. In 2015, the Centers for Disease Control and Prevention recorded 9,563 new cases of TB.

That same year, for the first time since 1992, the number of tuberculosis cases in the U.S. rose, according to the CDC. Twenty-nine states and the District of Columbia reported more cases in 2015 than they did in 2014. The per-capita rate of tuberculosis cases has plateaued at three infections per 100,000 people.

Read more: http://goo.gl/wSPDl4

First Case Of Sexual Transmission Of Zika Virus Reported

Lovers 5Published in LA WEEKLY BY DENNIS ROMERO

Zika’s a particularly evil little virus that could cause microcephaly, a rare neurological condition that causes affected infants to be born with abnormally small heads. This week the U.S. Centers for Disease Control announced a recent case of sexually transmitted Zika reported  in the Dallas area.

“According to a Dallas County Health Department investigation, a person who recently traveled to an area with Zika virus transmission returned to the United States and developed Zika-like symptoms,” the CDC said in a statement. “The person later tested positive for Zika, along with their sexual partner, who had not traveled to the area.”

That said, reports of sexually transmitted Zika are rare, and experts say the most common form of transmission is via mosquito bites in South America, particularly Brazil, as well as in the Caribbean, Central America, Mexico, Cape Verde and certain Pacific islands (American Samoa, Samoa, Tonga).

Health officials warned pregnant women to avoid or postpone travel to those areas.

The L.A. County Department of Public Health says pregnant women who have traveled to those regions and who have “symptoms suggestive of Zika virus infection during or within two weeks of travel” should get tested.

“The most important messages concern people who may be traveling to locations in the world where Zika virus outbreaks are currently occurring, and advising them on measures they need to take to protect their own health and prevent bringing the disease back here to Los Angeles County,” the county’s interim health officer, Dr. Jeffrey Gunzenhauser, said yesterday.

The CDC says avoiding sexual contact with potential Zika patients probably is wise.

“Based on what we know now, the best way to avoid Zika virus infection is to prevent mosquito bites AND to avoid exposure to semen from someone who has been exposed to Zika virus or has been ill from Zika virus infection,” the CDC says.

There has been one case of Zika reported in L.A. And given our pathways to Latin America, it shouldn’t surprise anyone if there are more. That case, reported in November, involved a girl who had traveled to El Salvador late last year and later recovered.

It sounds like you shouldn’t be too afraid. But you should definitely be aware. For the latest info on the virus, go here.

Heath Issues: 2 Million People Can’t Fight This Infection

Capsule23,000 People Die Each Year Because They Don’t Read Labels (Original article published by ‘UNITEDVOICE’

Does your family doctor prescribe an antibiotic when you or a family member get an infection? Doctors routinely use antibiotics as a first line of defense in fighting infections. The drugs work almost immediately to kill off the bacteria that’s causing the infection. Millions upon millions of lives are saved each year with these miracle drugs. Without them, you could easily die from what started out as a small infection in or on your body.

So, what’s the big concern? What if the antibiotics didn’t do their job in your body? What if you were one of the 2,000,000 people each year who can’t depend on antibiotics to do its job in killing infections? What if your were one of the 23,000 each year who die as a result of having developed a resistance to the miracle antibiotic drugs.

Why didn’t the antibiotics do its job?

ACCORDING TO A THREE YEAR STUDY BYCONSUMER REPORTS, AMERICANS ARE EATING FOODS THAT THAT ARE MAKING THEM RESISTANT TO CRITICAL ANTIBIOTIC DRUGS.

Low levels of antibiotics are routinely fed to healthy animals on a daily basis to promote growth and to kill bacteria that can result from conditions under which animals are raised. Over time, some bacteria survive and mutates within the animals to become a bacteria (superbug) that is resistant to antibiotics. When we eat these foods, we can ingest superbugs that are resistant to antibiotics, thus making us resistant to needed antibiotics when we get an infection.

What’s the solution? The Food and Drug (FDA) needs to promote stronger guidelines for raising food animals and farmers need to use better hygiene practices and growth management in raising animals for human consumption. The main problem foods are chicken, shrimp, ground beef, and turkey.

What can consumers do? We can buy foods labeled as Organic, Raised without Antibiotics, Certified Humane Animal Welfare, or American Grass Fed Certified. Finally, we can make sure we are handling uncooked foods properly and cooking it thoroughly.

http://www.unitedvoice.com/2-million-people-cant-fight-this-infection/

Ebola Beyond Sierra Leone: A Nightmare might be unfolding in Mano River Basin

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An Ebola victim being carried away in Liberia.

If anyone thinks the Ebola Outbreak in Mano River basin is something that is a trifle, please let me confidently state to them that the ongoing disaster is far from being something to put on back-burner. This is a time for all residents therein to unite and step-up our guard!
The World Health Organisation (WHO) Spokesperson Tarik Jasarevic has now officially informed journalists on what many had suspected was the fearful realisation that the new outbreak of Ebola in Liberia with an index case of a 17 years old schoolboy, was not really ‘new’ after all. The outbreak never really ended in Liberia.

WHO Spokesman has now confirmed that genetic studies of the virus in the ‘latest’ outbreak in Liberia is identical to the one that used to kill in Liberia few months back and which is the same virus that continues to kill in both Sierra Leone and Guinea.

However, none (I repeat, NONE) of the Liberians now with new Ebola infection in Liberia ever travelled to Sierra Leone or Guinea. It means the virus has been right inside Liberia quietly all this time.

The WHO Spokesman is now saying the infection of the 17 years old boy was likely acquired from a ‘non-identified transmission within the community’ or from ‘a survivor still carrying the infection in other body fluids long after the blood tested negative for the virus’.

There is also an other possibility (so fearful to contemplate) that the virus has now modified itself so much so that it can delay the onset of symptoms in those it infects.

What do I mean by ‘delay the onset of symptoms’? Let me explain. Viruses can exist for stated periods in humans before they start manifesting sickness in the infected human. For example, the HIV virus can exist for years in a person before it causes the manifestation of clinical signs of HIV-AIDS.

Now, prior to this Outbreak, Ebola was known to manifest symptoms within 2 to 21 days of infection. This particular MRU outbreak had an average of 9 days between infection and symptoms.

So, if, as is now suspected to be scenario for ‘new index case’ of a teenage boy in Liberia, this MRU Ebola virus is now with the ability to exist for more than 21 days in a human before it manifests symptoms of Ebola sickness, then I can easily say we have a major situation on our hands in the MRU basin.

Add to this, the huge number of survivors living in MRU basin as possible carriers of the dreadful Ebola virus in body fluids like male semen or in placenta of foetus in wombs, we might have a serious nightmare unfolding in the Mano River Union basin.

Honestly, this is the time to rally around our various leaderships and do whatever we can to give undiluted support to the three Presidents. In Sierra Leone, the country is still under a State of Emergency that was declared by President Koroma NOT to deliberately subdue our political discourse but to subdue Ebola.

Let us put aside Politics for a while and re-focus our attention on combating Ebola. The alternative to ending the Ebola Outbreak in MRU basin is too fearsome to contemplate…. Too fearful to imagine.

The writer

Koroma
The writer Dr. Sylvia Blyden and the president of Sierra Leone, Bah Koroma
The author is a trained medical doctor with an amazing versatility that makes her hold her own in many disciplines. Also a major Publisher and news journalist, Doctor Sylvia Blyden is a politician who has worked for the Sierra Leone Government as the first, and so far the only woman, to be ever appointed with Cabinet Rank to the Office of the President when she served as the Special Executive Assistant (SEA) to President Koroma for a period of two years.
           She gracefully resigned her position in October 2014. Doctor Sylvia Blyden, a member of the ruling All Peoples Congress (APC) of Sierra Leone, remains to be one of the most trusted allies of the Sierra Leone President and she was part of his presidential delegation to the UN High level Ebola Recovery Summit held from July 9th to 10th in New York during which two days period, she served as an Adviser to His Excellency the President.
             As far as the current Ebola Outbreak is concerned, Dr. Blyden is noted as the very first Sierra Leonean to raise an alarm in May 2014 over unexplained strange deaths in Kissi chiefdoms of Eastern Sierra Leone; the deaths turned out to be from Ebola – just as she had expressed suspicion. To date, the good Doctor has continued to be an irrepressible voice in the fight against Ebola.

 

The development Of Ebola Laboratory By Hitler’s Vassals In USA, Russia, Germany, Belgium And The Philippines.

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By Johan Van Dongen and Joel Savage

Western journalists, soldiers, politicians, as well as scientists, now over more than a decade after the second millennium, should realize what exactly took place in the last two hundred years on the African continent. Africa is used as a dumping ground for drugs, testing of drugs, exposing people to bio-warfare products and deliveries of war material to the wrong regimes, which have adversely affected the continent as well.

Actually you could say that the rest of the world have seriously abused Africa and degraded its people, as if they are creatures walking around on earth without brains, yet Africans are in the same products of Quantum Physics and Quantum Mechanics realized as whites, so I don’t understand why the many white leaders have taken advantage to treat blacks unfairly?

The aforementioned establishments should know what actually took place on the continent of Africa and therefore should also be aware of the fact that the United States Of  America, that claim ‘In God We Trust’  turned against God and became a land which supported Hitler’s evil and Nazi war criminals.

The shocking story of how America became one of the world’s safest postwar havens for Nazis, has revealed in Eric Lichtblau’s remarkable book: “The Nazis Next door. How America becomes a safe haven for Hitler’s men.”

Thousands of Nazis from concentration camps, guards to high-level officers in the Third Reich and other Nazi criminals, came to the United States after World War II and settled quietly to begin a new life. They had little trouble getting in, with scant scrutiny, many gained entry on their own as self-styled war “refugees,” avoiding the detection of their criminal history and their war crimes soon forgotten. But some had help and protection from the U.S. government.

The CIA, FBI, and the military, gave support to Hitler’s minions to work as spies, intelligence assets, and leading scientists and engineers, whitewashing their histories.

In the United States of America, the Nazi war criminals collaborated with top American scientists, to manufacture viruses of deadly diseases, financed by the CIA, the Rockefeller Foundation and their counterparts the Rothschilds, thus; Ebola and Aids viruses were some of the engineered diseases and spread by the Germans and Americans.

The same Nazi war criminals became normal American citizens, while years after the Second World War, the Jews captured in war concentration camps, were still going through the cruelties of life.  In America, the Nazi criminals taught students and they found out the way of making deadly viruses in animals. Financed by Bill Gates, vaccines were contaminated with the deadly viruses to be inflicted on Africans.

All the manufactured viruses, such as the Ebola virus got their names according to how they were prepared in the concerned laboratory or how it originated. But can someone for a moment think of and ask the reason Aids and Ebola have killed thousands of Africans.

*In 1989, the CDC reports, Ebola-Reston virus was introduced into quarantine facilities in Virginia and Pennsylvania by monkeys imported from the Philippines. No humans were infected.

*In 1990, Ebola-Reston virus was introduced once again into quarantine facilities in Virginia and Texas by monkeys imported from the Philippines. Four humans developed antibodies but did not get sick.

*In 1996, Ebola-Reston virus was introduced into a quarantine facility in Texas by monkeys imported from the Philippines. No human infections were identified.

*In May of 2004, a Russian scientist died of Ebola after accidentally pricking herself with a syringe while conducting research on infected guinea pigs in Siberia.

*A similar accident with Ebola had reportedly occurred several months earlier at the US Army’s biodefense laboratory at Fort Detrick in Frederick, Md., but the researcher involved didn’t acquire the disease. This incident is not listed on the CDC’s list of confirmed outbreaks, perhaps because the researcher didn’t develop antibodies.

In 2009, a scientist in Berlin, Germany accidentally pricked herself and was infected with Ebola. She was given an experimental vaccine as part of her treatment and did not become ill.

And only accidentally somebody dies because of a stupid accident. And what about all those students who are writing scientific papers and abstracts, as three of them are depicted, published in top medical journals in the last seventy years! Because Aids and Ebola viruses were man-made long before their outbreaks in black communities in Africa.

Lethal experimental infections of rhesus monkeys by aerosolized Ebola virus

Johnson E1, Jaax N, White J, Jahrling P,

Int J Exp Pathol. 1995 Aug;76(4):227-36.

Abstract

The potential of aerogenic infection by Ebola virus was established by using a head-only exposure aerosol system. Virus-containing droplets of 0.8-1.2 microns were generated and administered into the respiratory tract of rhesus monkeys via inhalation. Inhalation of viral doses as low as 400 plaque-forming units of virus caused a rapidly fatal disease in 4-5 days.

The illness was clinically identical to that reported for parenteral virus inoculation, except for the occurrence of subcutaneous and venipuncture site bleeding and serosanguineous nasal discharge. Immunocytochemistry revealed cell-associated Ebola virus antigens present in airway epithelium, alveolar pneumocytes, and macrophages in the lung and pulmonary lymph nodes; extracellular antigen was present on mucosal surfaces of the nose, oropharynx, and airways.

Aggregates of the characteristic filamentous virus were present within the type I pneumocytes, macrophages, and air spaces of the lung by electron microscopy. Demonstration of fatal aerosol transmission of this virus in monkeys reinforces the importance of taking appropriate precautions to prevent its potential aerosol transmission to humans.

Transmission of Ebola virus (Zaire strain) to uninfected control monkeys in a biocontainment laboratory

Jaax N1, Jahrling P, Geisbert T, Geisbert J, teele K, McKee K, Nagley D, Johnson E, Jaax G, Peters C.

Lancet. 1995 Dec 23-30;346(8991-8992):1669-71.

Abstract

Secondary transmission of Ebola virus infection in humans is known to be caused by direct contact with infected patients or body fluids. We report transmission of Ebola virus (Zaire strain) to two of three control rhesus monkeys (Macaca mulatta) that did not have direct contact with experimentally inoculated monkeys held in the same room.

The two control monkeys died from Ebola virus infections at 10 and 11 days after the last experimentally inoculated monkey had died. The most likely route of infection of the control monkeys was aerosol, oral or conjunctival exposure to virus-laden droplets secreted or excreted from the experimentally inoculated monkeys. These observations suggest approaches to the study of routes of transmission to and among humans.

Lethal experimental infection of rhesus monkeys with Ebola-Zaire (Mayinga) virus by the oral and conjunctival route of exposure

Davis K.J, Geisbert TJ, Vogel P, Jaax GP, Topper M, Jahrling PB.

Arch Pathol Lab Med. 1996 Feb;120(2):140-55.

Abstract

OBJECTIVE

The source of infection or mode of transmission of Ebola virus to human index cases of Ebola fever has not been established. Field observations in outbreaks of Ebola fever indicate that secondary transmission of Ebola virus is linked to improper needle hygiene, direct contact with infected tissue or fluid samples, and close contact with infected patients.

While it is presumed that the virus infects through either break in the skin or contact with mucous membranes, the only two routes of exposure that have been experimentally validated are parenteral inoculation and aerosol inhalation. Epidemiologic evidence suggests that aerosol exposure is not an important means of virus transmission in natural outbreaks of human Ebola fever; this study was designed to verify that Ebola virus could be effectively transmitted by oral or conjunctival exposure in nonhuman primates.

MATERIALS AND METHODS

Adult rhesus monkeys (Macaca mulatta) were exposed to Ebola-Zaire (Mayinga) virus orally (N=4), conjunctively (N=4), or by intramuscular inoculation (N=1, virus-positive control).

RESULTS

Four of seven monkeys exposed by the conjunctival route, three of four monkeys exposed by the oral route, and the intramuscularly inoculated positive control monkey were successfully infected with Ebola-Zaire (Mayinga). Seven monkeys died of Ebola fever between days 7 and 8 post-exposure, but one of the monkeys given aggressive supportive therapy and a platelet transfusion; lived until day 12 post-exposure.

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Belgian scientist and discoverer of Ebola, Peter Piot, knew everything about the virus but wouldn’t say publicly was a medical crime against Africa, because his country was involved.

CONCLUSIONS

Findings from the experiment study confirm that Ebola virus can be effectively transmitted via the oral or conjunctival route of exposure in nonhuman primates.

Well, Nazi students prepared many dangerous viruses, thus; everyone blames them for the crimes they committed, but the real question is: Who discovered officially those devilish and deadly viruses? It was a Belgian scientist named Peter Piot!

Peter Piot

Nearly 40 years ago, a young Belgian scientist traveled to a remote part of the Congolese rainforest – his task was to help find out why so many people were dying from an unknown and terrifying disease. In September 1976, a package containing a shiny blue thermos flask arrived at the Institute of Tropical Medicine in Antwerp, Belgium.

Working in the lab that day was Peter Piot, a 27-year-old scientist and medical school graduate training as a clinical microbiologist. “It was just a normal flask like any other you would use to keep coffee warm,” recalls Piot, now Director of the London School of Hygiene and Tropical Medicine. But this thermos wasn’t carrying coffee – altogether inside was a different cargo.

Nestled among a few melting ice cubes were vials of blood along with a note. It was a Belgian doctor based in what was then Zaire, now the Democratic Republic of Congo – his handwritten message explained that the blood was that of a nun, also from Belgium, who had fallen ill with a mysterious illness which he couldn’t identify.

The samples were treated like others, lab tested, but when the scientists placed some of the cells under an electron microscope they saw something they didn’t expect. “We saw a gigantic worm-like structure – gigantic by viral standards,” says Piot. It was a very unusual shape for a virus, only one other virus looked like that of the Marburg virus.” The Marburg virus was first discovered in 1967 when 31 people became ill with hemorrhagic fever in the cities of Marburg and Frankfurt in Germany and in Belgrade, the capital of Yugoslavia.

This Marburg outbreak was associated with laboratory staffs who were working with infected monkeys imported from Uganda – seven people died. Piot knew how serious Marburg could be – but after consulting experts around the world, he got confirmation that what he was seeing under the microscope wasn’t Marburg, but different which hasn’t been seen before. After that what’s next?Marburg

Malaria: Deadly Disease Still A Threat To Africa

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Malaria is a disease different from Acquired Immune Deficiency Syndrome-AIDS, but both have something in common, they don’t discriminate.

Malaria has been a long time tropical disease that has ravaged the African continent before the white explorers landed on the shores of Africa. It took many of them to their untimely grave; hence they referred the continent to ‘The white man’s grave.’

Despite the amazing discovery of technology, health care improvement and vaccines, malaria continues to kill hundreds of children and adults every year in Africa. The sickness is caused by a single-cell parasite called Plasmodium. Anopheles mosquitoes, usually females pick up the parasite from infected people when they bite. After bitten, the blood they obtained nurtures their eggs.

Inside the mosquito the parasites develop and reproduce. When the mosquito bites again, the parasites mix with its saliva and pass into the blood of the person being bitten. Africa’s fragile health care system and poverty have caused wide spread of the disease at a faster rate like the Acquired Immune Deficiency Syndrome (AIDS). Other factors which have escalated malaria in Africa, is the poor drainage system.

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Waste disposal, recycling and poor drainage systems, remain a key challenge facing every city in Africa. Stagnant pools, choked gutters and marshy places later become a breeding ground for mosquitoes where they lay their eggs. A malaria victim may show no symptoms for weeks after bitten by mosquitoes, until the parasites return to the bloodstream and invade the red blood cells.

Rapid multiplication of the parasites ruptures the red cells, releasing more parasites into the bloodstream and causing the characteristic symptoms. If the person does not receive prompt and effective drug therapy, damage may occur to the brain and other organs, sometimes leading to death. In many parts of Africa, where a sick person goes to the hospital if only he can afford, a malaria victim has no chance to recover than to succumb to the disease.

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The victim loses appetite, preventing the desire to eat. Weak and confined permanently to bed, malaria victim sleeps for hours. At times the victim sweats profusely and efforts to sleep become a nightmare. Malaria statistics indicate that over half a million (655, 000) people die from malaria each year, mostly children younger than five years old.

There are an estimated 216 million cases of malaria each year. Although the vast majority of malaria cases occur in sub-Saharan Africa, the disease is a public-health problem in more than 109 countries in the world, 45 of which are in Africa. Approximately 3.3 billion people live in areas where malaria is a constant threat. 90% of all malaria deaths occur in sub-Saharan Africa.

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Malaria eradication has been on the discussion table for years but still remains an illusion, since poverty is the source of all diseases in Africa. However, measures are applied to control the disease. Bed nets, domestic spraying insecticides, spraying infected places with DDT and anti—malaria vaccine help to protect people and the environment from malaria.

However; until the African government finds solution to its waste disposal problems and poor underground drainage systems, the possibility of eliminating or reducing malaria in the continent of Africa will be a dream of illusion.

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