Zika virus: Your questions answered

zika childBy Gretchen Vogel, Jon Cohen, Martin Enserin

Where did the Zika virus come from?

First isolated in 1947 and first described in a paper in 1952, Zika has long been known to occur in Africa and Southeast Asia—but until a decade ago, fewer than 15 cases had been described in the scientific literature. In 2007, the virus caused a big outbreak on Yap, an island group in the Western Pacific that is part of the Federated States of Micronesia; since then, it went on a major tour of other Pacific Islands before it landed in Brazil, from where it started spreading rapidly to other parts of South America, Central America, Mexico, and the Caribbean.

Why has it exploded so suddenly?

There may have been big outbreaks in Africa and Asia in the past that went undetected; scientists weren’t paying much attention. But the current massive epidemic was an event waiting to happen. Latin America has huge numbers of A. aegypti, also known as the yellow fever mosquito, an important vector for Zika. (The Asian tiger mosquito, A. albopictus, which is on the rise around the world, is believed to be a vector as well.) In addition, nobody in the Americas had immunity to the virus. Travel makes it worse. Aedes mosquitoes don’t fly more than a few hundred meters during their lives; Zika travels from city to city and country to country when infected people get on cars, buses, trains, and planes.

These combined factors meant that the virus had the ability to spread far and fast once it had arrived.

Will Zika spread to the United States and Europe?

Both the United States and Europe have already seen “imported cases”—people who arrived from a Zika-affected country carrying the virus. This was widely expected given the size of the epidemic in Latin America. The key question is whether there will be local outbreaks—that is, mosquitoes spreading the virus from person to person. There’s definitely a chance; A. albopictus occurs in several countries in southern Europe (and it may move north), while the southern and eastern United States have populations of both A. aegypti and A. albopictus.

If so, scientists expect outbreaks to be much smaller than elsewhere, based on past experience with mosquito-borne diseases. Recent dengue outbreaks in Florida, Texas, and Hawaii haven’t sickened more than a few hundred people, for instance; an outbreak of a mosquito-borne disease called chikungunya in northern Italy in 2007—which started when a man infected with the virus arrived from India—ended after 197 cases. One reason that outbreaks in these countries tend to be smaller may be that people spend less time outside and live in houses that are more difficult for mosquitoes to enter; mosquito population sizes may play a role as well.

Do we know for sure that Zika is causing a rise in birth defects?

No. There is strong circumstantial evidence that areas in Brazil hit hard by Zika have experienced a sharp increase in the number of babies born with microcephaly, a condition in which the head is much smaller than normal because the brain fails to develop properly. But it will take at least several months before the results from the first case-control studies of pregnant women infected with Zika are available. Doctors in Brazil first noticed an increase in cases of microcephaly during ultrasounds of pregnant women in June and July, a few months after the sudden rise in Zika infections. Fetal medicine expert Manoel Sarno, who works at the Federal University of Bahia, says the pattern of brain damage he is seeing now looks distinct from microcephaly caused by other infections, such as cytomegalovirus (CMV) or rubella. He and his colleagues started a study in August that is following women infected with Zika during their pregnancy; the results could come out late summer. Similar studies are underway elsewhere in Brazil and in Colombia.

Are there other urgent questions that scientists are asking?

Plenty. Scientists have difficulty determining who has been infected and who hasn’t because diagnostic tests have limitations. The most accurate tests—which detect viral RNA in a patient’s blood—only work within a week of the first symptoms appearing. After that time, researchers can test for antibodies in the blood. But current tests for Zika antibodies cross-react with antibodies to dengue, which is so widespread in Brazil—and much of the rest of Latin America—that almost all adults have antibodies to it. That makes it difficult to tell whether the mother of a baby born with microcephaly was infected with Zika earlier in her pregnancy.

Researchers would also like to know how often Zika is transmitted through sexual contact. One U.S. scientist who caught the virus in Africa passed it to his wife after he got home in 2008, and a second case of suspected sexual transmission happened in French Polynesia in 2013. But researchers have no idea what the risk is. (“If I was a man and I got Zika symptoms, I’d wait a couple of months before having unprotected sex,” virologist Scott Weaver of the University of Texas Medical Branch in Galveston recently told The New York Times.)

What drugs are available against Zika?

None. Until last year, Zika was so rare and believed to be so mild, that nobody bothered to look for candidate drugs. Even now that the virus is surging, it’s not obvious that there’s a big market for an antiviral drug, because the vast majority of those infected have very few symptoms or none at all. And it’s not clear that a drug could prevent birth defects when women contract Zika during pregnancy; by the time they become infected and develop symptoms, it may be too late to prevent such damage. A vaccine against Zika may offer more hope of preventing microcephaly.

And when can we expect a vaccine?

That will take years. Several groups have begun to make candidate Zika vaccines, a process that will take at least several months. Most of these vaccine approaches are piggybacking on existing vaccines. For example, many vaccines are made by stitching proteins from a pathogen’s surface into a harmless virus or vector; that is now being tried with Zika using those same vectors. Once a candidate vaccine is made, it will have to be tested in animals before humans.Human trials begin with small safety studies, then move on to larger studies that test whether the candidate product works. All of that usually takes 10 to 15 months. Given the urgency, the timeline could be compressed, but even so, Anthony Fauci, the director of the U.S. National Institute of Allergy and Infectious Diseases, told STAT that it may be at least 5 to 7 years before a Zika vaccine is commercially available.

Then what can we do to stop the spread of the virus?

Stop mosquitoes from biting people. Countries and communities can try to reduce mosquito populations by removing the small water reservoirs—such as flower pots, empty bottles, and discarded tires—in which Aedes mosquitoes like to breed. People can also reduce their personal exposure—especially important for women who are or might become pregnant—by putting screens on windows, covering their skin, and using insect repellant. However, history has shown that the impact of mosquito control on epidemics is modest at best, and they’re difficult to sustain.

There must be better ways to control mosquitoes?

Not yet but they’re in the works. A British biotech called Oxitec—which was recently purchased by Intrexon, a U.S. synthetic biology company—has developed A. aegyptimosquitoes containing a gene construct that will kill their offspring before they reach adulthood. When massive numbers of male individuals of this strain are released in the wild, they will mate with local females, producing offspring that are not viable, which has been shown to make a dent in the population.

In another line of research, scientists are infecting A. aegypti with a bacterium named Wolbachia, which reduces mosquitoes’ ability to transmit diseases. The researchers developing these approaches were mostly thinking about dengue, but Zika’s surge is giving their attempts a new sense of urgency. But again, it will take several years before these strategies are ready for prime time.

Bio-warfare Laboratories Of German And Japanese War Criminals Under The Guidance Of USA: The Revealing Voices of AIDS/HIV Theory Dissidents


By Johan van Dongen and Joel Savage

The horrific Aids pandemic, tremendously has generated scientific controversies within and outside the scientific establishment. Only a minority of scientists, like Johan van Dongen, and other engaged people have access to inside information concerning (bio-warfare) Aids and Ebola research.

As an experimental micro-surgeon in the early seventies, almost at the beginning of the multiple organ transplantation era,  Micro-Surgeon Johan van Dongen did carried out thousands of experimental organ transplantation. In order to deal with organ rejection, he administered, radiation and sera for diminishing the immunity of the organ receiver. Besides that he also administered uncountable agents to recipients of organs in order to trigger, diminish or completely wipe out the immune capacity which can be compared with Aids.

During his university and hospital appointments in the early seventies, and later undercover in the pharmaceutical industry, he discovered that animals didn’t die because of rejection of the transplanted organ but because of multiple infections which can be compared with human Aids victims. So, Johan van Dongen noticed that Aids can be induced by radiation, aflatoxins, Immuran/prednisolone combination, anti-lymphocyte sera, and many other bio-warfare agents.

Dormant HIV virus

As head of the Department of Experimental Microsurgery, and involved in all transplantation and immunological experiments, Johan also had been involved in many controversies. Especially the connection of his work and the polemic concerning the transmission of HIV.  In many ways, he discovered not only in his experiments but also in the extensive scientific literature the role of an obligatory co-factor that trans-activates the “Dormant” virus HIV in specific human cells. This obligatory co-factor which trans-activates the “Dormant” virus in specific human cells are deliberately introduced into mostly black-skinned people, collectively, Africans, governed by massive environmental factors, as you can read in our book: “Aids and Ebola the greatest crime in medical history against mankind,” in order to depopulate Africa.

Therefore we will always like to enlighten readers about the real origin of Aids and the true nature of famous international researchers as Robert Gallo. And as far as Gallo is concerned, Ricardo Veronesi, professor of the Faculty of Medicine at the University of Sao Paulo, was personally informed about the true nature of  Gallo’s research long before this controversy turned into a public scandal and as a consequence thousands of scientific Aids dissidents.

It was no less than Francoise Barré-Sinoussi of the French Pasteur Institute, who revealed the criminal intention of Gallo. And not only she became an Aids dissident but also the discovery of the HIV virus Luc Montagnier disputed Gallo, the fake discoverer of the HIV virus. In their opinion, the major bursts in the common scientific approach lie in its ignoring that the pathogenic of the HIV. Indeed it is governed by multiple deliberate environmental factors and one of these determinant factors is the PCR test (Polymerase Chain Reaction Test).

Polymerase Chain Reaction Test

This test is a technology in molecular biology used to amplify a single copy or a few copies of a piece of DNA across several orders of magnitude, generating thousands to millions of copies of a particular DNA sequence. Especially the diagnosis of hereditary diseases; the identification of genetic fingerprints, used in forensic sciences and paternity testing; and the detection and diagnosis of infectious diseases. The PCR test can be used to investigate the connection of diseases to the specific black race. Moreover, PCR can be extensively modified to perform a wide array of genetic manipulations not only in humans but also from microorganisms which cause Aids and Ebola.

Using an extrapolation of these kinds of techniques we can conclude that almost all persons who have HIV in their bodies, were purposely infected with this virus which can lead to Aids. Bio-warfare scientists are able to make black people artificially susceptible to HIV or Ebola by using controllable diseases as a cover-up.

Most of the biowarfare research using viruses which cause Aids and Ebola was predominantly carried out in Germany and Japan until 1945 and since then mainly in the USA and France, using Nazi and Japanese (military) scientific war criminals.

The Revealing Voices of Aids/HIV Theory Dissidents

The official scientific origin of the diverse HIV-strains has been placed somewhere between 1938 and 1948 when scientist T.F. Smith et al published an article in the authoritative medical journal Nature about this period in 1988 named: “The phylogenetic history of immunodeficiency virus”.

He wasn’t the only scientists who revealed the true nature of the HIV virus. Smith’s efforts to reveal the real origin of HIV was followed, to name a few, by Sharp et al with his article: “Understanding the origins of Aids viruses”, also in Nature, followed by Meyers et all with: “The phylogenetic analysis of the HIVs”. But the most important article is described in the top of the bill of medical journals the Lancet by scientist L.A. Evans et al who discovered the; “Simultaneous isolation of HIV-1 and HIV-2 from an Aids patient”.

All these mentioned scientists agreed that the distribution of the HIV virus was an intentional action. Their findings make it very conceivable that this distribution was intentional because sometimes both the new viruses HIV-1 and HIV-2, respectively HTLV-IV, are existing in one and the same person according to Evans. And because his publication is checked by the editing and scientific boards of the Lancet the outcome of his investigation was true. This counts also for thousands of publications in other medical journals as described in our book “Aids and Ebola the greatest crime in medical history against mankind.”

German scientist Wolff Geisler

According to the famous Aids/HIV theory dissident Wolff Geisler, further evidence of the intentional distribution, out of the mentioned simultaneous infection of the same persons, it was described as a second Aids epidemic in the same black-skinned population, by an inefficient transmission of the HIV virus. The appearance of this extremely rare retrovirus among the African Aids patients is so conspicuous that some world-famous scientists uttered a sentence about it. They alleged this to be; “Only another acquired opportunistic infection but rather an additional death sentence”. But is it?

In Africa, the probability of an early death of HIV patients is three times bigger than elsewhere when HIV patients are simultaneously infected with HTLV-1 as described in the Lancet by Page et al in his scientific publication: HTLV-I/II seropositivity and death from Aids among HIV-I seropositive intravenous drug users (Lancet, 1990; 335: 1439-41), an even more extremely important publication for the Aids/HIV theory dissidents. Because especially HTLV-I, among many other HIV viruses, was only demonstrated in Uganda, Ghana, South Africa, and Namibia.  HIV patients only in these countries appear simultaneously up till now.

According to Wolff Geisler, the concomitant existence of HTLV-I and HIV produces the observed rate of Aids patients in Uganda, Kenya and black-skinned people in Florida, USA and some Caribbean Islands, even though in general black people are by nature more resistant against HIV-infection than pale-skinned persons (see below). This means the HIV viruses are genetically engineered as described in our book.

No less than Luc Montagnier et al, the discoverer of the HIV virus stated that this virus is made out of the Nazi eugenics and genetic engineered experiments as well as the development of Aids-causing viruses in horses. In a very talked about an article he described in the authoritative Annals of Virology: “A new type of retrovirus from patients presenting with lymphadenopathy and acquired immune deficiency syndrome”: Structural and anti-genetic relatedness with Equine Infectious Anemia Virus EIAV (horse Aids), 1984; 135E: 119-31.

Equine Infectious Anemia Virus EIAV (HIV/Horse-Aids) made by Nazi Germany.

If we compare these findings to our references in “Aids the greatest crime in medical history against mankind” the book now available at Amazon, the HLA-A, B, C, DR3 and DR5 loci, is examined by the Nazi’s led by Otmar Verschuer.

In 1956 he joined the American Eugenics Society and worked under auspices of the Rockefeller-fund. He was also head of the Department of the Kaiser Wilhelm Institute in Germany.

Furthermore, we have to take into account that within people who have blood type HLA-DR3 Aids, it is much less common than in people who have the HLA-DR5 type. Under the Nazi’s research, it is important to note that precisely the HLA-DR5 type occurs mainly in Jews. The HLA-DR3 type contrast is most common in dark-colored Africans.

These two shreds of evidence or references are enough to let you know vividly what took place. In general, you can say that it is harder for blacks to get Aids than as it is for whites, but blacks have been made susceptible for a broad spectrum of brand new diseases caused by Germans, partly under the auspices of the South African Apartheid regime, and after the war under guidance of the U.S.A.

Nowadays we now know that monkeys do not get Aids when infected with the human Aids virus. The same goes for tuberculosis until the moment that monkeys in a laboratory made receptive. Therefore black-skinned people are under no circumstances contaminated with Aids by monkeys with or without eating them. That is so to speak a criminal scientific fairy tale.